[1] |
Guziewicz NA,Massetti AJ,Perez-Ramirez BJ,et al.Mechanisms of monoclonal antibody stabilization and release from silk biomaterials[J].Biomaterials,2013,34(31):7766-7775.
|
[2] |
Zhu Z,Wang D P,Ma Y,et al.Optimization of antioxidant silk fibroin peptide hydrolysis process[J].Adv Mater Res,2015,1073-1076:1789-1792.
|
[3] |
Yeo JH,Lee KG,Kweon HY,et al.Fractionation of a silk fibroin hydrolysate and its protective function of hydrogen peroxide toxicity[J].J Appl Polym Sci,2006,102(1):772-776.
|
[4] |
Park DS,Lee SH,Choi YJ,et al.Improving effect of silk peptides on the cognitive function of rats with aging brain facilitated by D-galactose[J].Biomol Ther,2011,19(2):224-230.
|
[5] |
Zhou F,Xue Z,Wang J.Antihypertensive effects of silk fibroin hydrolysate by alcalase and purification of an ACE inhibitory dipeptide[J].J Agric Food Chem,2010,58(11):6735-6740.
|
[6] |
Kang YK,Lee W,Kang B,et al.Memory-enhancing effects of silk fibroin-derived peptides in scopolamine-treated mice[J].J Microbiol Biotechnol,2013,23(12):1779-1784.
|
[7] |
Kim DK,Kang YK,Lee MY,et al.Neuroprotection and enhancement of learning and memory by BF-7[J].J Health Sci,2005,51(3):317-324.
|
[8] |
Lee MY,Lee SH,Lee JS,et al.BF-7 improved memory function and protected neuron from oxidative stress[J].Korean J Phys Anthrop,2004,17:313-320.
|
[9] |
Chen S,An FM,Yin L,et al.Glucagon-like peptide-1 protects hippocampal neurons against advanced glycation end product-induced tau hyperphosphorylation[J].Neuroscience,2014,256:137-146.
|
[10] |
Miklossy J.Bacterial amyloid and DNA are important constituents of senile plaques:further evidence of the spirochetal and biofilm nature of senile plaques[J].J Alzheimers Dis,2016,53(4):1459-1473.
|
[11] |
Tramutola A,Triplett JC,Di Domenico F,et al.Alteration of mTOR signaling occurs early in the progression of Alzheimer disease(AD):analysis of brain from subjects with pre-clinical AD,amnestic mild cognitive impairment and late-stage AD[J].J Neurochem,2015,133(5):739-749.
|
[12] |
Wang JZ,Xia YY,Grundke-Iqbal I,et al.Abnormal hyperphosphorylation of tau:sites,regulation,and molecular mechanism of neurofibrillary degeneration[J].J Alzheimers Dis,2013,33(s1):S123-S139.
|
[13] |
Martin L,Latypova X,Wilson C M,et al.Tau protein phosphatases in Alzheimer′s disease:the leading role of PP2A[J].Ageing Res Rev,2013,12(1):39-49.
|
[14] |
Llorens-Marítin M,Jurado J,Hernández F,et al.GSK-3β,a pivotal kinase in Alzheimer disease[J].Front Mol Neurosci,2014,7:46.
|
[15] |
Yu WP,Qian ZY,Xu GL,et al.Effects of crocetin on the myocardial cell damages due to oxidative stress[J].J China Pharm Univ(中国药科大学学报),2003,34(5):452-455.
|
[16] |
Butterfield DA,Swomley AM,Sultana R.Amyloid β-peptide(1-42)-induced oxidative stress in Alzheimer disease:importance in disease pathogenesis and progression[J].Antioxid Redox Signal,2013,19(8):823-835.
|