[1] |
. Curr Med Res Opin,2011,27(8):1635-1643.
|
[2] |
Bhatt JM,Lin HW,Bhattacharyya N. Prevalence,severity,exposures,and treatment patterns of tinnitus in the United States[J]. JAMA Otolaryngol Head Neck Surg,2016,142(10):959-965.
|
[3] |
Galazyuk A,Brozoski TJ. Animal models of tinnitus:a review[J]. Otolaryngol Clin North Am,2020,53(4):469-480.
|
[4] |
Li SH,Han DY,Yang WY,et al. Research progress on the ototoxicity mechanism of sodium salicylate[J]. J Audiol Speech Pathol(听力学及言语疾病杂志),2001,9(3):174-176.
|
[5] |
Steffens DC,Wei Jiang,Krishnan KR,et al. Metabolomic differences in heart failure patients with and without major depression[J]. J Geriatr Psychiatry Neurol,2010,23(2):138-146.
|
[6] |
Li WW,Yang Y,Wang SC,et al. Research progress of cellular metabolomics[J]. J Nanjing Univ Tradit Chin Med(南京中医药大学学报),2017,33(2):187-192.
|
[7] |
Lu Z,Li S,Sun R,et al. Hirsutella sinensis treatment shows protective effects on renal injury and metabolic modulation in db/db mice[J]. Evid Based Complement Alternat Med,2019,2019:4732858.
|
[8] |
Wang W,Cai Q,Zhou F,et al. Impaired pentose phosphate pathway in the development of 3D MCF-7 cells mediated intracellular redox disturbance and multi-cellular resistance without drug induction[J]. Redox Biol,2018,15:253-265.
|
[9] |
McEvilly M,Popelas C,Tremmel B. Use of uridine triacetate for the management of fluorouracil overdose[J]. Am J Health Syst Pharm,2011,68(19):1806-1809.
|
[10] |
Gallai V,Mazzotta G,Montesi S,et al. Effects of uridine in the treatment of diabetic neuropathy:an electrophysiological study[J]. Acta Neurol Scand,1992,86(1):3-7.
|
[11] |
Mironova GD,Khrenov MO,Talanov EY,et al. The role of mitochondrial KATP channel in anti-inflammatory effects of uridine in endotoxemic mice[J]. Arch Biochem Biophys,2018,654:70-76.
|
[12] |
Le TT,Ziemba A,Urasaki Y,et al. Disruption of uridine homeostasis links liver pyrimidine metabolism to lipid accumulation[J]. J Lipid Res,2013,54(4):1044-1057.
|
[13] |
Sun Q. Glutamate excitotoxicity and the effect of glutamate-glutamine cycle in the cochlea (耳蜗谷氨酸兴奋性毒性及谷氨酸—谷氨酰胺循环机制的研究)[D]. Beijing:PLA Postgraduate Medical School,2004.
|
[14] |
Fuhrman S,Palkovits M,Cassidy M,et al. The regional distribution of N-acetylaspartylglutamate (NAAG) and peptidase activity against NAAG in the rat nervous system[J]. J Neurochem,1994,62(1):275-281.
|
[15] |
Zhao J,Ramadan E,Cappiello M,et al. NAAG inhibits KCl-induced[3H]-GABA release via mGluR3,cAMP,PKA and L-type calcium conductance[J]. Eur J Neurosci,2001,13(2):340-346.
|
[16] |
Sanabria ER,Wozniak KM,Slusher BS,et al. GCP II (NAALADase) inhibition suppresses mossy fiber-CA3 synaptic neurotransmission by a presynaptic mechanism[J]. J Neurophysiol,2004,91(1):182-193.
|
[17] |
Romei C,Raiteri M,Raiteri L. Glycine release is regulated by metabotropic glutamate receptors sensitive to mGluR2/3 ligands and activated by N-acetylaspartylglutamate (NAAG)[J]. Neuropharmacology,2013,66:311-316.
|
[18] |
Fulop T,Radabaugh S,Smith C. Activity-dependent differential transmitter release in mouse adrenal chromaffin cells[J]. J Neurosci,2005,25(32):7324-7332.
|