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缺血预处理对大鼠四动脉结扎后氧化应激及脑水肿的保护作 …[J]. 中国药科大学学报, 2000, (5): 55-58.
引用本文: 缺血预处理对大鼠四动脉结扎后氧化应激及脑水肿的保护作 …[J]. 中国药科大学学报, 2000, (5): 55-58.
Ischemic Preconditioning is More Effective to Scavenge Oxidative Stress and Cerebral Edema Against Cerebral Infarct Size by Four-vessel Occlusion in Rats[J]. Journal of China Pharmaceutical University, 2000, (5): 55-58.
Citation: Ischemic Preconditioning is More Effective to Scavenge Oxidative Stress and Cerebral Edema Against Cerebral Infarct Size by Four-vessel Occlusion in Rats[J]. Journal of China Pharmaceutical University, 2000, (5): 55-58.

缺血预处理对大鼠四动脉结扎后氧化应激及脑水肿的保护作 …

Ischemic Preconditioning is More Effective to Scavenge Oxidative Stress and Cerebral Edema Against Cerebral Infarct Size by Four-vessel Occlusion in Rats

  • 摘要: 观察缺血预处理对大鼠四动脉结扎法的全脑缺血/再灌注损伤的保护作用。预处理后,缺血30min,再灌注10及30min。记录并比较EEG、MDA、脑含水量和脑梗塞范围。脑缺血期间脑电波变平,缺血预处理减少脑电波的变化,并且减少梗塞范围。再灌注10和30min后,对梗塞抑制率分别为20%和54%。缺血预处理也可将MDA和含水量恢复到正常水平。预处理消除氧化应激反应和脑水肿,比减少脑梗塞范围更有效。

     

    Abstract: To investigate the protective effect of preconditioning on global cerebral ischemic damage by 4-vessel occlusion model in rats. Preconditioning was established followed by 30 min ischemia and reperfusion for 10 and 30 min, separately. Data of EEG ampli

     

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